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Heritable pulmonary arterial hypertension (HPAH) is an autosomal dominant disorder with incomplete penetrance characterized by progressive pulmonary vascular obstruction and right ventricular failure. Variants in the TBX4 gene have been identified in pediatric PAH cohorts at frequencies of 10–30%, implicating TBX4 as a predisposing gene in HPAH (PMID:27770446, PMID:28877973).
Functional studies demonstrate that TBX4 variants disrupt nuclear localization and DNA-binding, leading to impaired transcriptional activation of FGF10 and abnormal lung morphogenesis in ex vivo models, consistent with pulmonary vascular pathology (PMID:35914404).
Key take-home: TBX4 testing is clinically useful for early detection and risk stratification in families with heritable PAH.
Gene–Disease AssociationLimitedVariants in TBX4 identified in pediatric PAH cohorts (10–30% of cases) with incomplete penetrance ([PMID:27770446], [PMID:28877973]) Genetic EvidenceLimitedTBX4 variants reported in cohort studies of pediatric PAH (10–30% frequency) without segregation data Functional EvidenceModerateIn vitro assays show impaired TBX4-mediated FGF10 activation and ex vivo lung explant morphogenesis defects ([PMID:35914404]) |