Variant Synonymizer: Platform to identify mutations defined in different ways is available now!
Over 2,000 gene–disease validation summaries are now available—no login required!
Bi-allelic variants in BRWD1 have been identified in three unrelated individuals with autosomal recessive primary ciliary dyskinesia (PCD) and multiple morphological abnormalities of the sperm flagella (MMAF). Exome sequencing of 53 patients with MMAF plus respiratory symptoms uncovered two homozygous missense variants and one compound-heterozygous missense variant in BRWD1, including c.5573A>T (p.Gln1858Leu) (PMID:33389130). All three unrelated probands (PMID:33389130) exhibited chronic sinusitis (HP:0011109) and bronchiectasis (HP:0002110), consistent with PCD, with no additional segregating affected relatives reported.
Functional studies demonstrated absence of BRWD1 protein in patient respiratory cilia and sperm flagella, with transmission electron microscopy revealing loss of inner and outer dynein arms in both tissues and microtubule doublet defects in respiratory cilia. A mouse Brwd1 loss-of-function model recapitulated oligoasthenoteratospermia and infertility, supporting a loss-of-function mechanism in ciliogenesis and spermiogenesis (PMID:33389130; PMID:18353305).
Key Take-home: BRWD1 should be included in genetic testing panels for autosomal recessive PCD and related ciliopathies.
Gene–Disease AssociationLimitedThree unrelated probands (PMID:33389130) with bi-allelic BRWD1 variants and concordant functional data Genetic EvidenceModerateIdentification of biallelic missense variants in three probands with PCD-like respiratory and sperm flagellar phenotypes (PMID:33389130) Functional EvidenceModerateAbsence of BRWD1 in patient cilia with dynein arm defects, and Brwd1 mouse model recapitulates spermiogenesis and ciliary dysfunction (PMID:33389130; PMID:18353305) |