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MED27Neurodevelopmental Disorder with Spasticity, Cataracts, and Cerebellar Hypoplasia

MED27-related neurodevelopmental disorder is an autosomal recessive condition characterized by global developmental delay, cognitive impairment, limb spasticity, cataracts, and cerebellar hypoplasia. To date, two pedigrees have been reported: a single patient with a homozygous c.74G>A (p.Arg25His) variant (PMID:39296199) and a consanguineous family with two affected siblings harboring c.839C>T (p.Pro280Leu) (one additional affected relative) (PMID:35876425). In vitro functional studies of Arg25His demonstrated significantly reduced MED27 protein expression and weakened MED27–MED14 interaction (PMID:39296199). No conflicting reports have been described. These data support a Limited clinical validity classification for MED27 in NEDSCAC.

Key Take-home: MED27 sequencing should be considered in patients with early-onset developmental delay, spasticity, cataracts, and cerebellar hypoplasia.

References

  • Heliyon • 2024 • Novel homozygous missense variants in MED27 associated with neurodevelopmental disorder: Clinical and pathogenetic research. PMID:39296199
  • Movement Disorders • 2022 • MED27, SLC6A7, and MPPE1 Variants in a Complex Neurodevelopmental Disorder with Severe Dystonia. PMID:35876425

Evidence Based Scoring (AI generated)

Gene–Disease Association

Limited

Two pedigrees (one singleton, one sib pair), minimal segregation beyond sibs, concordant functional data

Genetic Evidence

Limited

Homozygous missense variants in two families with AR inheritance and segregation in one sib pair

Functional Evidence

Moderate

In vitro assays show reduced MED27 protein and disrupted MED27–MED14 interaction