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Nevoid Basal Cell Carcinoma Syndrome (NBCCS) is an autosomal dominant tumor predisposition syndrome most often caused by heterozygous PTCH1 mutations. A single case report described a 13-year-old girl with multiple keratocystic odontogenic tumors and rib anomalies carrying a heterozygous PTCH2 frameshift variant, c.1172_1173del (p.Phe390_Ser391insTer), supporting a potential causative role for PTCH2 in NBCCS despite milder phenotypic presentation (PMID:23479190). However, no additional affected relatives were reported, and a healthy individual homozygous for a PTCH2 frameshift (c.269del (p.Gly90AlafsTer4)) and her heterozygous daughter show no NBCCS features, calling into question pathogenicity of PTCH2 loss-of-function alleles (PMID:30820324).
Functional assays have demonstrated that a PTCH2 missense variant, c.2156G>A (p.Arg719Gln), abolishes PTCH2 inhibitory activity on Hedgehog signaling in cell‐based reporter assays (PMID:18285427), and distinct PTCH2 splice isoforms modulate SHH‐N internalization without fully recapitulating PTCH1 function (PMID:14613484). However, a cohort of 21 PTCH1/SUFU-negative GS families yielded no pathogenic PTCH2 variants, and population data reveal homozygous PTCH2 loss-of-function alleles in healthy individuals, refuting a major role for PTCH2 in NBCCS predisposition (PMID:34170463).
Key Take-home: Current genetic and population evidence refute PTCH2 as a primary NBCCS gene; PTCH2 should be excluded from clinical diagnostic panels for Gorlin syndrome.
Gene–Disease AssociationRefutedNo pathogenic PTCH2 variants identified in 21 PTCH1/SUFU-negative GS families and presence of healthy homozygous LoF individual; conflicting case reports ([PMID:34170463]; [PMID:30820324]) Genetic EvidenceLimitedSingle proband with PTCH2 frameshift reported without segregation; healthy homozygotes and absence in cohort studies ([PMID:23479190]; [PMID:30820324]; [PMID:34170463]) Functional EvidenceLimitedIn vitro loss of PTCH2 inhibitory activity for p.Arg719Gln and differential HH pathway regulation by splice isoforms ([PMID:18285427]; [PMID:14613484]) |